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发表于 2010-11-27 22:32:58
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ARGUMENT53
Thirteen years ago, researchers studied a group of 25 infants who showed signs of mild distress when exposed to unfamiliar stimuli such as an unusual odor or a tape recording of an unknown voice. They discovered that these infants were more likely than other infants to have been conceived in early autumn, a time when their mothers' production of melatonin-a hormone known to affect some brain functions-would naturally increase in response to decreased daylight. In a follow-up study conducted earlier this year, more than half of these children-now teenagers-who had shown signs
of distress identified themselves as shy. Clearly, increased levels of melatonin before birth cause shyness during infancy and this shyness continues into later life.
提纲:
1,在早秋怀孕与母亲激素的分泌没有关系;婴儿的害羞是先天遗传, 不受激素影响。
2,没有证据表明青少年时期的害羞是在婴儿时期形成的;
3,调查样本过少,不具有代表性,研究者可能特意选择了有症状的婴儿,即使婴儿有代表性, 研究者只研究了两种刺激,忽视了其它刺激的影响;
3,总结,应该提供婴儿害羞与母亲m分泌的证据及婴儿害羞影响以后生活的证据;
字数:443
正文:
In this argument, the speaker concludes that increased levels of melatonin before birth can cause shyness during infancy and this infancy continues into later life. To support his conclusion, the speaker cites a follow-up study of twenty five infants in which the infants showed signs of distress when exposed to certain unfamiliar stimuli. The speaker also points out that those infants who identified themselves as shy then they grew up to teenagers thirteen years later were probably conceived in early autumn when their mothers’ production of melatonin was at an increased level. However, I found this argument logically flawed in several critical respects.
First of all, the speaker bases his conclusion on the unsound assumption that it is the mother's production of melatonin that determines whether a infant is shy. Though early autumn is a time when a mother is at her high level of melatonin production, it does not mean that it is the very reason that an infant gets effected. Moreover, a mother's level of hormone production and her health condition has nothing to do with a baby's shyness, which is a congenital characteristic inherited from its parents. The merely fact of early autumn-born infant hardly proves his conclusion.
Secondly, no evidence shows that those teenagers who identified themselves as shy were shy when they were newly born. Besides the influence of their parents' character, teenagers’ character are also effected by the environment they are brought up, maybe those shy teenagers live in a relatively confined space and have less communication with their friends than others, or what is worse, some of them suffered domestic violence during their childhood. So, other than some serious diseases, sign of shyness showed during infancy barely continues into later life.
Thirdly, the follow-up study is unconvincing for several reasons. The speaker does not provide evidence that the infants studied were randomly chosen, it is possible that the researchers chose the infants with a sign of distress particularly despite of other normal infants. Even if these twenty five infants are representative, the researchers only included such types of stimuli as an unusual odor and a tape recording of unknown voice ignoring the other possible stimuli. Moreover, sign of mild distress does not indicate the shyness of a baby, so the assumption of shyness during infancy is unfounded.
In sum, the argument is unconvincing as it stands. To strengthen it, the speaker must provide clear evidence that increased level of a mother's production of melatonin can cause her baby to be shy, and that shyness during infancy continues into later life. To better evaluate the argument we would need more information about the study for its confirmation. |
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